7 Household Items for Faster Fat Loss




1. Apple Cider Vinegar:


According to a recent paper published in the Journal of the  Academy of Nutrition and Dietetics, vinegar has been purported to be a weight-loss aid since the early 1800s. Research has shown that vinegar may promote weight loss by suppressing appetite, delaying gastric emptying, or exerting a “carb blocking” effect by inhibiting certain carbohydrate-digesting enzymes.


Perhaps the most noteworthy benefits of apple cider vinegar relate to its effects on carbohydrate management. Recent research even suggests that an individuals glycemic response may be a key variable in determining the best diet for that person.
Even more, emerging evidence suggests that the better ones carb tolerance, the more carbs that s/he can eat while still losing fat.4–6 Research has also shown that poor insulin sensitivity, carbohydrate intolerance, and poor glycemic control can lead to weight gain, reduce energy levels, increase appetite, increase cravings, and more.


Vinegar has been shown to possess numerous benefits for insulin function and glycemic control. In one study, researchers from Arizona State University found that participants’ insulin sensitivity improved by as much as 34% when they consumed apple cider
vinegar (about four teaspoons) before a carbohydrate-containing meal compared to when they ate the same meal without apple cider vinegar.


In a randomized crossover trial published in the European Journal of Clinical Nutrition, researchers found that consuming apple cider vinegar before eating white bread (providing 50 grams of carbohydrate) reduced participants’ glycemic response by 31% compared to when the white bread was eaten without vinegar.


Numerous additional studies have fortified the ability of apple cider vinegar (as little as two teaspoons) to significantly improve insulin sensitivity and reduce the glycemic response when taken before a carbohydrate-containing meal.


In addition to its effects on carbohydrate management, apple cider vinegar has also been shown to reduce hunger, increase satiety, and ultimately, lead to reduced calorie intake at subsequent meals. In a randomized, crossover trial, researchers found that

consuming apple cider vinegar before a carbohydrate-containing meal led to not only a substantial improvement in acute glycemic control, it also led to participants eating 250
fewer calories over the rest of the day.


In a separate study published in the European Journal of Clinical Nutrition, Swedish researchers found that consuming vinegar prior to a carbohydrate-containing meal led to significant increases in satiety (or feelings of fullness and satisfaction). The level of satiety was proportionate to the amount of vinegar consumed.

Naturally, with beneficial acute effects on carbohydrate management and satiety, apple cider vinegar has the potential to be a useful tool to promote weight management. In one randomized, double-blind, placebo-controlled trial, Japanese researchers found that participants who ingested apple cider vinegar daily for 12 weeks lost significantly more body weight, belly fat, and inches from their waistlines compared to the placebo group. Interestingly, folks who consumed two tablespoons of apple cider vinegar today showed greater improvements in these areas than those who took one tablespoon.



JUST IN 14 DAYS

VANISHED 6 POUNDS FAT 

Download free E-book





2. BAKING SODA:

Also known as sodium bicarbonate, perhaps the most well-known “non-household” use for baking soda is for athletic and exercise performance. In fact, it’s been used as an “ergogenic aid” (i.e., performance- enhancing) for over 80 years. 

Baking soda is considered to be “alkaline,” which means that it has a pH above 7.0 (i.e., neutral). Along these lines, any pH below 7.0 is considered to be acidic. The human body functions best when its acid-alkaline (or, acid-base) balance is at or near neutral. Generally speaking, the pH of the blood is around 7.4 while it’s around 7.0 in muscles. 

During exercise—particularly anaerobic exercise at higher intensities—one of the by- products of energy metabolism is a compound called lactic acid, which is then split into lactate and a hydrogen ion. This build-up of hydrogen ions (not lactic acid itself) results in a decrease in pH (i.e., muscular acidosis), which is speculated to contribute to muscle fatigue. 

An elevation of bicarbonate may have a performance-enhancing effect by “buffering” hydrogen ions associated with intense activity.19 Specifically, sodium bicarbonate appears to increase the rate at which hydrogen ions are removed from the working muscles, which contributes to the maintenance of intramuscular pH. 

In a systemic review with meta-analysis (a high-level statistical analysis of all studies on a given topic) published in the journal Sports Medicine, Australian researchers confirmed that consumption of sodium bicarbonate (0.3 g/kg body mass) prior to exercise significantly improved performance in a one-minute all-out bout of exercise. 

In general, studies show that the beneficial effects of baking soda on performance are relegated to activities and exercise lasting 1 – 7 minutes, and it may also be useful for sports and activities involving intermittent or sustained periods of high-intensity work. 

In addition, sodium bicarbonate may also improve resistance training performance. In a randomized, double-blind, crossover study published in the Journal of Strength and Conditioning Research, researchers from Coventry University found that ingestion of sodium bicarbonate before a strength training session led to a significantly greater number of reps performed and repetitions to failure in the squat and bench press in resistance-trained men. 

How does all of this relate to fatloss? Simple. On one hand, increased exercise performance (e.g., exercising at a higher intensity, doing more exercise in a given time, performing more work at a given intensity) all lead to an increase in calorie expenditure. Along those lines, in order to lose weight, one must burn more calories than s/he eats (i.e., negative energy balance) on a regular, consistent basis—a fundamental principle
of metabolism and thermodynamics.


With regard to improving strength training, it is the most powerful tool for building and maintaining muscle mass, and adding exercise to a weight loss program tends to help spare muscle mass. This is key because muscle mass comprises the most metabolically active tissues of the body, and losses in calorie-burning muscle mass when dieting may predispose one to weight regain. 

This is why a key objective during weight loss is to reduce body fat while minimizing loss of FFM to promote optimal overall health, metabolic function, cardiovascular health, and physical functioning. This is why an emphasis should be placed on fat loss as opposed
to weight loss. 

In general, high-intensity interval training (HIT, i.e., brief repeated “bursts” of vigorous exercise interspersed with low-intensity recovery intervals), high-intensity resistance training (HIRT), and resistance training significantly increase metabolic rate, calorie burn (i.e., excess post-exercise oxygen consumption, EPOC), and fat burning in the hours (even days) after exercise.

These 5 steps reveal the things you absolutely MUST AVOID if you want to slow the aging process, reclaim your health, and achieve your ideal body.


CLICK HERE TO READ MORE






3. Cayenne Pepper:


Cayenne peppers contain a compound called capsaicin, and observational studies suggest that consumption of foods containing capsaicin is associated with lower body weight. Research has shown that capsaicin can boost metabolic rate, accelerate fat burning, decrease fat storage, increase feelings of fullness, reduce appetite, and decrease calorie intake. 

One unique way that capsaicin appears to increase its thermogenic effects (i.e., increased calorie burn) is through activation of “brown fat,” which is different from unwanted body fat (also called white adipose tissue).33 The purpose of brown fat is
to burn off calories as heat in order to keep the body warm, and capsaicin’s ability to “activate” stubborn brown fat and increase calorie burn is a cutting-edge way to boost metabolism and burn body fat.

In a study published in the International Journal of Obesity, Canadian researchers described several additional means by which capsaicin augments the impact of caloric restriction on weight loss. They reported that capsaicin has the potential to reduce— or completely prevent—the normal increase in hunger and decrease in fullness that typically accompanies a reduced-calorie diet. Furthermore, they provide evidence that capsaicin can combat the decrease in calorie expenditure and fat burning that typically

In a systemic review and meta-analysis (which is a high-level statistical analysis of all studies published on a specific topic) published in the journal Appetite, researchers from the United Kingdom found that consumption of capsaicin prior to a meal led to a significant decrease in calorie intake during the subsequent meal. Thus, capsaicin has the potential to promote fat loss both by boosting the metabolism (i.e., more calories burned) and reducing food intake and suppressing appetite (i.e., fewer calories consumed).
Not only that, capsaicin appears to activate a compound in the body called AMPK, which helps partition carbs to muscle, not fat. Activation of AMPK also increases fat burning and reduces the body’s ability to create and store fat.40 In other words, increasing AMPK activity means shuttling carbs to muscles to be used for fuel, burn more fat, and store less.

With all of that in mind, you might consider spicing up your meals a bit with cayenne. Other peppers, including jalapeños and habaneros also contain capsaicin. Basically, the hotter the pepper, the greater the capsaicin content, and if you’re feeling especially spicy, then you might try the bhut jolokia pepper (aka, the “ghost pepper”), which is so hot that it is often used as an elephant repellant in India.






4. Cinnamon:

Studies consistently show that whole cinnamon powder (provided in amounts ranging from 1 – 6 grams) is markedly effective at acutely improving carbohydrate tolerance and individual glycemic response.


In one randomized, double-blind, placebo-controlled study, participants were either given a standardized cinnamon extract or a placebo twice daily for two months. At the conclusion of the study, the participants taking the cinnamon extract experienced an improvement in carbohydrate management that was nearly five times greater than that of the placebo, and their post-meal glycemic response (after a carbohydrate-containing meal) also decreased an impressive 12%, which was four times greater than the placebo group.



In another randomized, double-blind, placebo-controlled study, participants were either given a standardized cinnamon extract or a placebo twice daily for 12 weeks. The participants taking the cinnamon extract significantly improved carbohydrate tolerance, and even though this wasn’t a weight loss study, they significantly improved body composition (i.e., decreased body fat, increased lean body mass).


In one study, healthy lean young men took 5 grams of cinnamon powder (about two teaspoons) or a placebo before undergoing an oral glucose tolerance test (OGTT), which involves drinking 75 grams of high-glycemic carbs. After taking the cinnamon, the participants significantly improved their glycemic response and demonstrated improved insulin sensitivity. What’s more, when the participants took the cinnamon 12 hours before the OGTT, they still experienced the same significant benefits in glycemic control, suggesting that the benefits of cinnamon appear to be both immediate and sustained for up to 12 hours.


Another one of the ways that cinnamon exerts its effects is by inhibiting the activity of certain carbohydrate-digesting enzymes (e.g., alpha-amylase, alpha-glucosidase). By inhibiting these enzymes, cinnamon has the potential to reduce or slow down the digestion of dietary carbohydrates (i.e., carb “blocking” effects). In a 2014 randomized, double-blind, placebo-controlled study, French researchers found that participants taking a cinnamon extract decreased post-meal blood sugar by 21% after 60 minutes and 15% after 2 hours (compared to the placebo group).47 They found that this improvement in carbohydrate metabolism was achieved without additional insulin secretion, supporting the notion that cinnamon “seems to specifically inhibit” carbohydrate-digesting enzymes (e.g., alpha-amylase).


In addition to its impact on glycemic control and carbohydrate management, cinnamon may also exert additional anti-obesity effects. In a study published in the journal Scientific Reports, researchers from Switzerland found that certain compounds in cinnamon may increase fat burning and reduce production of the ghrelin.


Ghrelin is often referred to as the “hunger hormone.” It is produced in the GI tract and functions as a signaling compound in the nervous system stimulating hunger.51 Ghrelin is released when the stomach is empty, and levels go down after eating.52 In addition
to stimulating hunger, ghrelin also “turns on” reward centers in the brain, increasing the pleasure and reward response to eating and reinforcing the consumption of rewarding, tasty food.



It’s important to note that there are numerous forms available. Although short-term trials have not demonstrated any adverse outcomes with Cinnamon cassia use, its high coumarin content is a concern during prolonged use. Coumarins are naturally- occurring plant compounds that are considered to be moderately toxic.57 Thus, while Cinnamon cassia has been used in numerous human trials (and shown to be both safe and effective), when purchasing cinnamon powder (e.g., from the grocery store), “true” cinnamon (i.e., Ceylon Cinnamon, Cinnamon verum, or Cinnamon Zeylanicum) is likely a better option, as its coumarin content is negligible.




5. Cumin:  

Cumin (Cuminum cyminum) is a flowering plant in the Apiacae family. Its seeds are used in a number of cuisines worldwide, both in whole and ground form. Cumin is widely used in Ayurvedic medicine to promote healthy digestion and detoxification, and it has been shown to possess antimicrobial, anti-inflammatory, and antioxidant properties.58 Multiple studies suggest that cumin has the potential to augment weight loss.


In a recent randomized, double-blind, placebo-controlled study published in the journal Complementary Therapies in Clinical Practice, researchers from Iran set out to assess the effects of cumin powder on body composition in healthy participants.59 In the study,
88 overweight and obese women were randomly assigned to one of two groups. Both groups of women consumed yogurt twice daily at meals for 3 months, with the experimental group having 3 grams of cumin (slightly more than one teaspoon) added to their yogurt. Both groups of women received the same nutritional counseling with appropriate guidance for weight loss. In other words, all women followed a supervised calorie-restricted diet.


At the end of the 3-month study, both groups of women lost weight, a result to be expected given the reduced-calorie diets both followed. However, the women who ate the yogurt plus cumin dropped more inches from their waists (49% more) and lost substantially more weight (89% more) and fat mass (70%) compared to the placebo group. While there was no statistically significant change in calorie-burning lean mass (i.e., neither group lost a significant amount, which is impressive in its own right), the cumin group dropped body fat percentage nearly 200% more than the placebo group.


In addition, the women consuming cumin also showed significantly greater improvements in blood triglycerides and blood cholesterol compared to the placebo group, including a 14-fold greater reduction in triglycerides, a 5-fold greater decrease in LDL cholesterol, and a 12-fold greater increase in HDL cholesterol.


In another randomized, double-blind, placebo-controlled trial published in the journal Annals of Nutrition and Metabolism, a separate group of Iranian researchers set out to determine the effects of supplementation with cumin on weight loss and metabolic profiles in overweight men and women


After 8 weeks, the cumin group (who took one 100mg cumin capsule three times daily) lost over 7 TIMES more weight than the placebo group. Perhaps even more interesting is that the cumin group experienced a similar decrease in weight and body mass index as a third group of participants, who took a prescription weight loss drug during the trial. Food journals were recorded throughout the duration of the study, and there were no differences in food intake (e.g., calories, carbs, fats). Even more, only the cumin group experienced significant improvements in insulin sensitivity.


6. Green Tea:  

According to researchers, Camellia sinensis, which is the plant species whose leaves and buds are used in the production of tea, exerts several “anti-obesity effects.”61 Although various types of teas (e.g., oolong, black, green) all come from the Camellia sinensis plant, green tea leaves are processed (i.e., fermented) differently, which leaves them with a higher concentration of beneficial polyphenols called catechins. It’s these compounds, which also have noteworthy anti- inflammatory and antioxidant properties, that seem to have quite a potent effect on the metabolism and fat burning, and what’s more, they may also suppress appetite and decrease the absorption of calories.


Studies consistently show that green tea extract (standardized for the catechin epigallocatechin gallate, EGCG) increases the body’s use of fat for fuel, and these effects are independent of caffeine/stimulants. It does so by inhibiting enzymes that can    shut down important fat-burning hormones (i.e., norepinephrine), thereby stoking the bodys fat-burning furnace.


In one study measuring 24-hour calorie expenditure and fat burning, healthy men supplementing with a green tea extract providing 90mg EGCG three times daily experienced a 4% increase in metabolic rate and 3.4% decrease in respiratory exchange ratio (RER), which signifies that they were burning more fat to meet the increased demand in calories.65 The participants taking the green tea extract were even burning more fat during sleep, and overall, over 41% of their daily calories burned came from fat—31% more fat burned than the placebo group.


In another study, participants taking a green tea extract daily (providing 400mg of EGCG ) for 4 weeks showed a 25% increase in fat burning, and what’s more, they lost over 1.5% body fat during the one-month trial.66 In a 12-week study, participants taking a green tea extract (providing 270mg of EGCG daily) experienced a 3.3% increase in metabolic rate, a 4.6% decrease in body weight, and a 4.5% reduction in waist circumference.
  
In yet another study, participants taking a green tea extract standardized for EGCG combined with a modest reduced-calorie diet lost over twice as much weight as the placebo group after just 8 weeks. The participants taking the green tea extract also experienced a 2-fold greater increase in metabolic rate compared to the placebo group.
In other words, green tea can help make a good fat loss program even more effective.
  
In addition to increasing metabolism, calorie expenditure, and fat burning, researchers also suggest that green tea extract can help control energy balance by suppressing appetite. In rats, researchers from the University of Chicago found that injections of
EGCG significantly reduced food intake and body weight. In a human study published in the International Journal of Obesity, participants consumed 8% fewer calories at a meal 4 hours after taking a standardized green tea extract when compared to the placebo group. Not only did the folks taking the green tea extract eat less, they also experienced a significant increase in metabolic rate, an effect that was maintained over the course of the 8-week trial and was accompanied by a significant reduction in body fat.

Researchers suggest consumption of 2 – 4 cups (i.e., 500mL – 1L) of green tea per day to reap these fat-burning and health promoting benefits.



7. Extra Vigin Olive Oil:  

Over the past several decades, a Mediterranean Diet has been promoted as effective tool to support heart health, as this type of diet has been shown to lower the risk of cardiovascular disease and many of its risk factors, including carbohydrate management, blood pressure, and lipid levels (e.g., cholesterol, triglycerides)


The Mediterranean Diet is characterized by up to 40% of its calories coming from fat, of which nearly half is of the monounsaturated variety. Of course, extra-virgin olive oil is a cornerstone of the Mediterranean Diet, which includes as many as eight servings of olive oil daily. Despite what you’ve been led to believe, dietary fat does not make you fat.


In fact, the Mediterranean Diet has also been reported to be protective against obesity, and sure enough, numerous studies have shown that it is an effective tool for weight management.


Like cinnamon, research has shown that olive oil may promote insulin sensitivity and glycemic control. A recent study that appeared in the journal Clinical Nutrition concluded that a higher intake of olive oil led to improved insulin sensitivity. Other studies documenting the health attributes of a Mediterranean Diet have alluded to the beneficial effects of olive oil on carbohydrate management.


Olive oil is rich in a specific monounsaturated fatty acid called oleic acid, which seems to have a potent impact on appetite regulation. Researchers from the University of California Irvine found that oleic acid stimulates the production of a compound called oleoylethanolamide (OEA) by the cells of the small intestine. OEA helps to suppress appetite by activating specific sites in the brain that help to curb hunger. Previously, this group of researchers found that increasing OEA levels reduces appetite, increases weight loss, and improves various metabolic parameters.


In a study published in the journal Food & Function, Italian researchers assessed whether the oleic acid content of a meal impacted OEA levels and calorie intake in healthy human volunteers. Participants consumed either two tablespoons of extra-virgin

olive oil (75% oleic acid), high-oleic sunflower oil (79% oleic acid), or sunflower oil (33% oleic acid) on three separate occasions. The researchers then measured calorie intake at a subsequent lunch and over the course of 24 hours.


In the two hours after lunch, only the extra-virgin olive oil condition resulted in reduced hunger and greater fullness and satisfaction. What’s more, when the participants consumed the extra-virgin olive oil, they ate over 250 fewer calories than when they consumed the sunflower oil with a lower oleic acid content. The researchers attributed this to the significant increase in OEA levels that were only noted after extra-virgin olive oil and high-oleic sunflower oil consumption.


Olive oil is naturally abundant in a polyphenol called oleocanthal, which has been shown to promote a healthy inflammatory response. Specifically, oleocanthal acts as a “natural anti-inflammatory compound” by inhibiting the activity of cyclooxygenase (COX) enzymes, a property it shares with the highly-recognizable NSAID ibuprofen.


In addition to oleocanthal, there are a number of other polyphenols (e.g., oleuropein, hydroxytyrosol) in extra-virgin olive oil that provide antioxidant activity and promote a healthy inflammatory response.84 In fact, Italian researchers have found that oleuropein, a compound that is similar in structure to oleocanthal, exerts anti-inflammatory effects.85
While fat tissue itself secretes inflammatory chemicals called cytokines, research has also shown that certain markers of persistent, unhealthy levels of inflammation may predict future weight gain.



REFERENCE:


1.     Heath C, Heath D. Switch: How to Change Things When Change Is Hard. 1st ed.

New York: Broadway Books; 2010.

2.      Kohn JB. Is vinegar an effective treatment for glycemic control or weight loss? J Acad Nutr Diet. 2015;115(7):1188. doi:10.1016/j.jand.2015.05.010.
3.     Zeevi D, Korem T, Zmora N, et al. Personalized Nutrition by Prediction of Glycemic

Responses. Cell. 2015;163(5):1079-1094. doi:10.1016/j.cell.2015.11.001.

4.      McClain AD, Otten JJ, Hekler EB, Gardner CD. Adherence to a low-fat vs. low- carbohydrate diet differs by insulin resistance status: Research Letter. Diabetes Obes Metab. 2013;15(1):87-90. doi:10.1111/j.1463-1326.2012.01668.x.
5.      Pittas AG, Roberts SB. Dietary Composition and Weight Loss: Can We Individualize Dietary Prescriptions According to Insulin Sensitivity or Secretion Status? Nutr Rev. 2006;64(10):435-448. doi:10.1111/j.1753-4887.2006.tb00174.x.
6.      Gardner CD, Offringa LC, Hartle JC, Kapphahn K, Cherin R. Weight loss on low-fat vs. low-carbohydrate diets by insulin resistance status among overweight adults and adults with obesity: A randomized pilot trial: Weight Loss-Diet and Insulin Resistance. Obesity. 2016;24(1):79-86. doi:10.1002/oby.21331.
7.     Nathan DM, Davidson MB, DeFronzo RA, et al. Impaired Fasting Glucose and

Impaired Glucose Tolerance: Implications for care. Diabetes Care. 2007;30(3):753-

759. doi:10.2337/dc07-9920.

8.     Grundy SM. Pre-Diabetes, Metabolic Syndrome, and Cardiovascular Risk. J Am

Coll Cardiol. 2012;59(7):635-643. doi:10.1016/j.jacc.2011.08.080.

9.      Riccardi G, Rivellese AA. Dietary treatment of the metabolic syndrome--the optimal diet. Br J Nutr. 2000;83 Suppl 1:S143-S148.
10.    Johnston CS, Kim CM, Buller AJ. Vinegar Improves Insulin Sensitivity to a High- Carbohydrate Meal in Subjects With Insulin Resistance or Type 2 Diabetes. Diabetes Care. 2004;27(1):281-282. doi:10.2337/diacare.27.1.281.
11.   Brighenti F, Castellani G, Benini L, et al. Effect of neutralized and native vinegar on blood glucose and acetate responses to a mixed meal in healthy subjects. Eur J Clin Nutr. 1995;49(4):242-247.
12.   Johnston CS, Steplewska I, Long CA, Harris LN, Ryals RH. Examination
of the antiglycemic properties of vinegar in healthy adults. Ann Nutr Metab.

2010;56(1):74-79. doi:10.1159/000272133.

13.    Liljeberg H, Björck I. Delayed gastric emptying rate may explain improved glycaemia in healthy subjects to a starchy meal with added vinegar. Eur J Clin Nutr. 1998;52(5):368-371.
14.    Leeman M, Ostman E, Björck I. Vinegar dressing and cold storage of potatoes lowers postprandial glycaemic and insulinaemic responses in healthy subjects. Eur J Clin Nutr. 2005;59(11):1266-1271. doi:10.1038/sj.ejcn.1602238.
15.    Johnston CS, Buller AJ. Vinegar and peanut products as complementary foods to reduce postprandial glycemia. J Am Diet Assoc. 2005;105(12):1939-1942. doi:10.1016/j.jada.2005.07.012.
16.    Ostman E, Granfeldt Y, Persson L, Björck I. Vinegar supplementation lowers glucose and insulin responses and increases satiety after a bread meal in healthy subjects. Eur J Clin Nutr. 2005;59(9):983-988. doi:10.1038/sj.ejcn.1602197.
17.    Kondo T, Kishi M, Fushimi T, Ugajin S, Kaga T. Vinegar intake reduces body weight, body fat mass, and serum triglyceride levels in obese Japanese subjects. Biosci Biotechnol Biochem. 2009;73(8):1837-1843.
18.    Debold EP, Beck SE, Warshaw DM. Effect of low pH on single skeletal muscle myosin mechanics and kinetics. AJP Cell Physiol. 2008;295(1):C173-C179. doi:10.1152/ajpcell.00172.2008.
19.   Lancha Junior AH, Painelli V de S, Saunders B, Artioli GG. Nutritional Strategies

to Modulate Intracellular and Extracellular Buffering Capacity During High-Intensity

Exercise. Sports Med Auckl NZ. 2015;45 Suppl 1:S71-S81. doi:10.1007/s40279-

015-0397-5.

20.   Carr AJ, Hopkins WG, Gore CJ. Effects of acute alkalosis and acidosis on performance: a meta-analysis. Sports Med Auckl NZ. 2011;41(10):801-814. doi:10.2165/11591440-000000000-00000.
21.   Burke LM. Practical considerations for bicarbonate loading and sports performance. Nestlé Nutr Inst Workshop Ser. 2013;75:15-26. doi:10.1159/000345814.
22.   Duncan MJ, Weldon A, Price MJ. The effect of sodium bicarbonate ingestion on back squat and bench press exercise to failure. J Strength Cond Res Natl Strength Cond Assoc. 2014;28(5):1358-1366. doi:10.1519/JSC.0000000000000277.
23.   Hall KD, Heymsfield SB, Kemnitz JW, Klein S, Schoeller DA, Speakman JR.

Energy balance and its components: implications for body weight regulation. Am J
Clin Nutr. 2012;95(4):989-994. doi:10.3945/ajcn.112.036350.

24.   Garrow JS. Exercise in the treatment of obesity: a marginal contribution. Int J Obes

Relat Metab Disord J Int Assoc Study Obes. 1995;19 Suppl 4:S126-S129.

25.    Redman LM, Heilbronn LK, Martin CK, et al. Effect of calorie restriction with or without exercise on body composition and fat distribution. J Clin Endocrinol Metab.
2007;92(3):865-872. doi:10.1210/jc.2006-2184.

26.   Stiegler P, Cunliffe A. The role of diet and exercise for the maintenance of fat- free mass and resting metabolic rate during weight loss. Sports Med Auckl NZ.
2006;36(3):239-262.

27.   Müller MJ, Bosy-Westphal A, Kutzner D, Heller M. Metabolically active components of fat-free mass and resting energy expenditure in humans: recent lessons from imaging technologies. Obes Rev Off J Int Assoc Study Obes. 2002;3(2):113-122.
28.   Faria SL, Kelly E, Faria OP. Energy expenditure and weight regain in patients submitted to Roux-en-Y gastric bypass. Obes Surg. 2009;19(7):856-859. doi:10.1007/s11695-009-9842-6.
29.   Gibala MJ, McGee SL. Metabolic Adaptations to Short-term High-Intensity Interval Training: A Little Pain for a Lot of Gain? Exerc Sport Sci Rev. 2008;36(2):58-63. doi:10.1097/JES.0b013e318168ec1f.
30.   Paoli A, Moro T, Marcolin G, et al. High-Intensity Interval Resistance Training (HIRT) influences resting energy expenditure and respiratory ratio in non-dieting individuals. J Transl Med. 2012;10(1):237. doi:10.1186/1479-5876-10-237.
31.   Whiting S, Derbyshire E, Tiwari BK. Capsaicinoids and capsinoids. A potential role for weight management? A systematic review of the evidence. Appetite.
2012;59(2):341-348. doi:10.1016/j.appet.2012.05.015.

32.   Leung FW. Capsaicin as an anti-obesity drug. Prog Drug Res Fortschritte

Arzneimittelforschung Prog Rech Pharm. 2014;68:171-179.

33.   Saito M, Yoneshiro T. Capsinoids and related food ingredients activating brown fat thermogenesis and reducing body fat in humans. Curr Opin Lipidol. 2013;24(1):71-
77. doi:10.1097/MOL.0b013e32835a4f40.

34.   Chechi K, Nedergaard J, Richard D. Brown adipose tissue as an anti-obesity

tissue in humans: Brown fat in humans. Obes Rev. 2014;15(2):92-106. doi:10.1111/

obr.12116.

35.   Carey AL, Kingwell BA. Brown adipose tissue in humans: Therapeutic

potential to combat obesity. Pharmacol Ther. 2013;140(1):26-33. doi:10.1016/j. pharmthera.2013.05.009.
36.    Tremblay A, Arguin H, Panahi S. Capsaicinoids: a spicy solution to the management of obesity? Int J Obes 2005. 2016;40(8):1198-1204. doi:10.1038/ ijo.2015.253.
37.    Whiting S, Derbyshire EJ, Tiwari B. Could capsaicinoids help to support weight management? A systematic review and meta-analysis of energy intake data. Appetite. 2014;73:183-188. doi:10.1016/j.appet.2013.11.005.
38.    Hwang J-T, Park I-J, Shin J-I, et al. Genistein, EGCG, and capsaicin inhibit adipocyte differentiation process via activating AMP-activated protein kinase. Biochem Biophys Res Commun. 2005;338(2):694-699. doi:10.1016/j. bbrc.2005.09.195.
39.    Kurth-Kraczek EJ, Hirshman MF, Goodyear LJ, Winder WW. 5’ AMP-activated protein kinase activation causes GLUT4 translocation in skeletal muscle. Diabetes.
1999;48(8):1667-1671.

40.    O’Neill HM, Holloway GP, Steinberg GR. AMPK regulation of fatty acid metabolism and mitochondrial biogenesis: implications for obesity. Mol Cell Endocrinol.
2013;366(2):135-151. doi:10.1016/j.mce.2012.06.019.

41.   Khan A, Safdar M, Ali Khan MM, Khattak KN, Anderson RA. Cinnamon

Improves Glucose and Lipids of People With Type 2 Diabetes. Diabetes Care.

2003;26(12):3215-3218. doi:10.2337/diacare.26.12.3215.

42.    Allen RW, Schwartzman E, Baker WL, Coleman CI, Phung OJ. Cinnamon Use in Type 2 Diabetes: An Updated Systematic Review and Meta-Analysis. Ann Fam Med. 2013;11(5):452-459. doi:10.1370/afm.1517.
43.    Davis PA, Yokoyama W. Cinnamon intake lowers fasting blood glucose: meta- analysis. J Med Food. 2011;14(9):884-889. doi:10.1089/jmf.2010.0180.
44.    Anderson RA, Zhan Z, Luo R, et al. Cinnamon extract lowers glucose, insulin and cholesterol in people with elevated serum glucose. J Tradit Complement Med. April
2015. doi:10.1016/j.jtcme.2015.03.005.

45.   Ziegenfuss TN, Hofheins JE, Mendel RW, Landis J, Anderson RA. Effects of

a Water-Soluble Cinnamon Extract on Body Composition and Features of the

Metabolic Syndrome in Pre-Diabetic Men and Women. J Int Soc Sports Nutr.

2006;3(2):45. doi:10.1186/1550-2783-3-2-45.

46.    Solomon TPJ, Blannin AK. Effects of short-term cinnamon ingestion on in vivo glucose tolerance. Diabetes Obes Metab. 2007;9(6):895-901. doi:10.1111/j.1463-
1326.2006.00694.x.

47.   Beejmohun V, Peytavy-Izard M, Mignon C, et al. Acute effect of Ceylon cinnamon
extract on postprandial glycemia: alpha-amylase inhibition, starch tolerance test in rats, and randomized crossover clinical trial in healthy volunteers. BMC Complement Altern Med. 2014;14(1):351. doi:10.1186/1472-6882-14-351.
48.    Mohamed Sham Shihabudeen H, Hansi Priscilla D, Thirumurugan K. Cinnamon extract inhibits α-glucosidase activity and dampens postprandial glucose excursion in diabetic rats. Nutr Metab. 2011;8(1):46. doi:10.1186/1743-7075-8-46.
49.   Adisakwattana S, Lerdsuwankij O, Poputtachai U, Minipun A, Suparpprom C.

Inhibitory activity of cinnamon bark species and their combination effect with acarbose against intestinal α-glucosidase and pancreatic α-amylase. Plant Foods Hum Nutr Dordr Neth. 2011;66(2):143-148. doi:10.1007/s11130-011-0226-4.
50.    Camacho S, Michlig S, de Senarclens-Bezençon C, et al. Anti-obesity and anti- hyperglycemic effects of cinnamaldehyde via altered ghrelin secretion and functional impact on food intake and gastric emptying. Sci Rep. 2015;5:7919. doi:10.1038/srep07919.
51.    Inui A, Asakawa A, Bowers CY, et al. Ghrelin, appetite, and gastric motility: the emerging role of the stomach as an endocrine organ. FASEB J Off Publ Fed Am Soc Exp Biol. 2004;18(3):439-456. doi:10.1096/fj.03-0641rev.
52.    Schwartz MW, Woods SC, Porte D, Seeley RJ, Baskin DG. Central nervous system control of food intake. Nature. 2000;404(6778):661-671. doi:10.1038/35007534.
53.    Dickson SL, Egecioglu E, Landgren S, Skibicka KP, Engel JA, Jerlhag E. The role of the central ghrelin system in reward from food and chemical drugs. Mol Cell Endocrinol. 2011;340(1):80-87. doi:10.1016/j.mce.2011.02.017.
54.    Egecioglu E, Jerlhag E, Salomé N, et al. Ghrelin increases intake of rewarding food in rodents. Addict Biol. 2010;15(3):304-311. doi:10.1111/j.1369-1600.2010.00216.x.
55.   Skibicka KP, Hansson C, Egecioglu E, Dickson SL. Role of ghrelin in food reward:

impact of ghrelin on sucrose self-administration and mesolimbic dopamine and acetylcholine receptor gene expression. Addict Biol. 2012;17(1):95-107. doi:10.1111/j.1369-1600.2010.00294.x.
56.    Wang Y-H, Avula B, Nanayakkara NPD, Zhao J, Khan IA. Cassia Cinnamon as a Source of Coumarin in Cinnamon-Flavored Food and Food Supplements in the United States. J Agric Food Chem. 2013;61(18):4470-4476. doi:10.1021/ jf4005862.
57.   Abraham K, Wöhrlin F, Lindtner O, Heinemeyer G, Lampen A. Toxicology

and risk assessment of coumarin: Focus on human data. Mol Nutr Food Res.
2010;54(2):228-239. doi:10.1002/mnfr.200900281.

58.    Al-Snafi AE. The pharmacological activities of Cuminum cyminum-A review. IOSR J Pharm. 2016;6(6):46-65.
59.    Zare R, Heshmati F, Fallahzadeh H, Nadjarzadeh A. Effect of cumin powder on body composition and lipid profile in overweight and obese women. Complement Ther Clin Pract. 2014;20(4):297-301. doi:10.1016/j.ctcp.2014.10.001.
60.    Taghizadeh M, Memarzadeh MR, Asemi Z, Esmaillzadeh A. Effect of the cumin cyminum L. Intake on Weight Loss, Metabolic Profiles and Biomarkers of Oxidative Stress in Overweight Subjects: A Randomized Double-Blind Placebo-Controlled Clinical Trial. Ann Nutr Metab. 2015;66(2-3):117-124. doi:10.1159/000373896.
61.    Torres-Fuentes C, Schellekens H, Dinan TG, Cryan JF. A natural solution for obesity: Bioactives for the prevention and treatment of weight gain. A review. Nutr Neurosci. 2015;18(2):49-65. doi:10.1179/1476830513Y.0000000099.
62.    Hursel R, Viechtbauer W, Westerterp-Plantenga MS. The effects of green tea on weight loss and weight maintenance: a meta-analysis. Int J Obes 2005.
2009;33(9):956-961. doi:10.1038/ijo.2009.135.

63.    Rains TM, Agarwal S, Maki KC. Antiobesity effects of green tea catechins: a mechanistic review. J Nutr Biochem. 2011;22(1):1-7. doi:10.1016/j. jnutbio.2010.06.006.
64.    Shixian Q, VanCrey B, Shi J, Kakuda Y, Jiang Y. Green tea extract thermogenesis- induced weight loss by epigallocatechin gallate inhibition of catechol-O- methyltransferase. J Med Food. 2006;9(4):451-458. doi:10.1089/jmf.2006.9.451.
65.    Dulloo AG, Duret C, Rohrer D, et al. Efficacy of a green tea extract rich in catechin polyphenols and caffeine in increasing 24-h energy expenditure and fat oxidation in humans. Am J Clin Nutr. 1999;70(6):1040-1045.
66.    Roberts JD, Roberts MG, Tarpey MD, Weekes JC, Thomas CH. The effect of a decaffeinated green tea extract formula on fat oxidation, body composition and exercise performance. J Int Soc Sports Nutr. 2015;12(1):1. doi:10.1186/s12970-
014-0062-7.

67.    Chantre P, Lairon D. Recent findings of green tea extract AR25 (Exolise) and its activity for the treatment of obesity. Phytomedicine Int J Phytother Phytopharm.
2002;9(1):3-8. doi:10.1078/0944-7113-00078.

68.    Auvichayapat P, Prapochanung M, Tunkamnerdthai O, et al. Effectiveness of green tea on weight reduction in obese Thais: A randomized, controlled trial. Physiol Behav. 2008;93(3):486-491. doi:10.1016/j.physbeh.2007.10.009.
69.    Kao YH, Hiipakka RA, Liao S. Modulation of endocrine systems and food intake by green tea epigallocatechin gallate. Endocrinology. 2000;141(3):980-987. doi:10.1210/endo.141.3.7368.
70.    Belza A, Frandsen E, Kondrup J. Body fat loss achieved by stimulation of thermogenesis by a combination of bioactive food ingredients: a placebo- controlled, double-blind 8-week intervention in obese subjects. Int J Obes 2005.
2007;31(1):121-130. doi:10.1038/sj.ijo.0803351.

71.    Kao YH, Hiipakka RA, Liao S. Modulation of obesity by a green tea catechin. Am J Clin Nutr. 2000;72(5):1232-1234.
72.    Liyanage T, Ninomiya T, Wang A, et al. Effects of the Mediterranean Diet on Cardiovascular Outcomes—A Systematic Review and Meta-Analysis. Wright JM, ed. PLOS ONE. 2016;11(8):e0159252. doi:10.1371/journal.pone.0159252.
73.   Davis C, Bryan J, Hodgson J, Murphy K. Definition of the Mediterranean Diet; a

Literature Review. Nutrients. 2015;7(11):9139-9153. doi:10.3390/nu7115459.

74.   Willett WC, Leibel RL. Dietary fat is not a major determinant of body fat. Am J Med.

2002;113 Suppl 9B:47S - 59S.

75.    Gotsis E, Anagnostis P, Mariolis A, Vlachou A, Katsiki N, Karagiannis A. Health benefits of the Mediterranean Diet: an update of research over the last 5 years. Angiology. 2015;66(4):304-318. doi:10.1177/0003319714532169.
76.    Mancini JG, Filion KB, Atallah R, Eisenberg MJ. Systematic Review of the Mediterranean Diet for Long-Term Weight Loss. Am J Med. 2016;129(4):407-415. e4. doi:10.1016/j.amjmed.2015.11.028.
77.    Sala-Vila A, Cofán M, Mateo-Gallego R, et al. Inverse association between serum phospholipid oleic acid and insulin resistance in subjects with primary dyslipidaemia. Clin Nutr Edinb Scotl. 2011;30(5):590-592. doi:10.1016/j. clnu.2011.02.008.
78.    Estruch R, Martínez-González MA, Corella D, et al. Effects of a Mediterranean- style diet on cardiovascular risk factors: a randomized trial. Ann Intern Med.
2006;145(1):1-11.

79.    Schwartz GJ, Fu J, Astarita G, et al. The lipid messenger OEA links dietary fat intake to satiety. Cell Metab. 2008;8(4):281-288. doi:10.1016/j.cmet.2008.08.005.
80.    Lo Verme J, Gaetani S, Fu J, Oveisi F, Burton K, Piomelli D. Regulation of food intake by oleoylethanolamide. Cell Mol Life Sci CMLS. 2005;62(6):708-716. doi:10.1007/s00018-004-4494-0.
81.   Mennella I, Savarese M, Ferracane R, Sacchi R, Vitaglione P. Oleic acid content
of a meal promotes oleoylethanolamide response and reduces subsequent energy intake in humans. Food Funct. 2015;6(1):204-210. doi:10.1039/c4fo00697f.
82.    Beauchamp GK, Keast RSJ, Morel D, et al. Phytochemistry: ibuprofen-like activity in extra-virgin olive oil. Nature. 2005;437(7055):45-46. doi:10.1038/437045a.
83.    Vane JR, Botting RM. New insights into the mode of action of anti-inflammatory drugs. Inflamm Res Off J Eur Histamine Res Soc Al. 1995;44(1):1-10.
84.    Bulotta S, Celano M, Lepore SM, Montalcini T, Pujia A, Russo D. Beneficial effects of the olive oil phenolic components oleuropein and hydroxytyrosol: focus on protection against cardiovascular and metabolic diseases. J Transl Med.
2014;12:219. doi:10.1186/s12967-014-0219-9.

85.    Impellizzeri D, Esposito E, Mazzon E, et al. Oleuropein aglycone, an olive oil compound, ameliorates development of arthritis caused by injection of collagen type II in mice. J Pharmacol Exp Ther. 2011;339(3):859-869. doi:10.1124/ jpet.111.182808.
86.    Engstrom G, Hedblad B, Stavenow L, Lind P, Janzon L, Lindgarde F. Inflammation- Sensitive Plasma Proteins Are Associated With Future Weight Gain. Diabetes.
2003;52(8):2097-2101. doi:10.2337/diabetes.52.8.2097.

87.   Keophiphath M, Priem F, Jacquemond-Collet I, Clément K, Lacasa D.

1,2-vinyldithiin from garlic inhibits differentiation and inflammation of human preadipocytes. J Nutr. 2009;139(11):2055-2060. doi:10.3945/jn.109.105452.
88.    Homayoni Rad A, Akbarzadeh F, Mehrabany EV. Which are more important: Prebiotics or probiotics? Nutrition. 2012;28(11-12):1196-1197. doi:10.1016/j. nut.2012.03.017.
89.    Costabile A, Kolida S, Klinder A, et al. A double-blind, placebo-controlled, cross- over study to establish the bifidogenic effect of a very-long-chain inulin extracted from globe artichoke (Cynara scolymus) in healthy human subjects. Br J Nutr.
2010;104(7):1007-1017. doi:10.1017/S0007114510001571.

90.   Kelly G. Inulin-type prebiotics--a review: part 1. Altern Med Rev J Clin Ther.

2008;13(4):315-329.

91.    Sleeth ML, Thompson EL, Ford HE, Zac-Varghese SEK, Frost G. Free fatty acid receptor 2 and nutrient sensing: a proposed role for fibre, fermentable carbohydrates and short-chain fatty acids in appetite regulation. Nutr Res Rev.
2010;23(01):135-145. doi:10.1017/S0954422410000089.

92.    Byrne CS, Chambers ES, Morrison DJ, Frost G. The role of short chain fatty acids in appetite regulation and energy homeostasis. Int J Obes. 2015;39(9):1331-1338.
doi:10.1038/ijo.2015.84.

93.    Guess ND, Dornhorst A, Oliver N, Bell JD, Thomas EL, Frost GS. A randomized controlled trial: the effect of inulin on weight management and ectopic fat in subjects with prediabetes. Nutr Metab. 2015;12:36. doi:10.1186/s12986-015-0033-2.





Comments

Total Pageviews

Popular Posts